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dc.contributor.authorRagone, Isabella
dc.contributor.authorBarallobre-Barreiro, Javier
dc.contributor.authorTakov, Kaloyan
dc.contributor.authorTheofilatos, Konstantinos
dc.contributor.authorYin, Xiaoke
dc.contributor.authorSchmidt, Lukas Emanuel
dc.contributor.authorDoménech, Nieves
dc.contributor.authorCrespo-Leiro, María Generosa
dc.contributor.authorvan der Voorn, Stephanie M.
dc.contributor.authorVink, Aryan
dc.contributor.authorvan Veen, Toon A.B.
dc.contributor.authorBödör, Csaba
dc.contributor.authorMerkely, Béla
dc.contributor.authorRadovits, Tamás
dc.contributor.authorMayr, Manuel
dc.date.accessioned2024-06-19T11:10:45Z
dc.date.available2024-06-19T11:10:45Z
dc.date.issued2023-08-14
dc.identifier.citationRagone I, Barallobre-Barreiro J, Takov K, Theofilatos K, Yin X, Schmidt LE, Domenech N, Crespo-Leiro MG, van der Voorn SM, Vink A, van Veen TAB, Bödör C, Merkely B, Radovits T, Mayr M. SERCA2a protein levels are unaltered in human heart failure. Circulation. 2023 Aug 15;148(7):613-616.es_ES
dc.identifier.issn0009-7322
dc.identifier.urihttp://hdl.handle.net/2183/37140
dc.descriptionResearch letteres_ES
dc.language.isoenges_ES
dc.publisherAmerican Heart Association (AHA)es_ES
dc.relation.urihttps://doi.org/10.1161/CIRCULATIONAHA.123.064513es_ES
dc.rightsCreative Commons Attribution 4.0 International License (CC-BY 4.0)es_ES
dc.rights.urihttp://creativecommons.org/licenses/by/4.0/*
dc.titleSERCA2a protein levels are unaltered in human heart failurees_ES
dc.typeinfo:eu-repo/semantics/articlees_ES
dc.rights.accessinfo:eu-repo/semantics/openAccesses_ES
UDC.journalTitleCirculationes_ES
UDC.volume148es_ES
UDC.issue7es_ES
UDC.startPage613es_ES
UDC.endPage616es_ES
dc.identifier.doi10.1161/CIRCULATIONAHA.123.064513


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