• Cardiac Protection Induced by Urocortin-2 Enables the Regulation of Apoptosis and Fibrosis After Ischemia and Reperfusion Involving miR-29a Modulation 

      Mayoral-González, Isabel; Calderón-Sánchez, Eva M.; Galeano-Otero, Isabel; Martín-Bórnez, Marta; Gutiérrez-Carretero, Encarnación; Fernández-Velasco, María; Doménech, Nieves; Crespo-Leiro, María Generosa; Gómez, Ana María; Ordoñez-Fernández, Antonio; Hmadcha, Abdelkrim; Smani, Tarik (Elsevier, 2022-01-28)
      [Abstract] Urocortin-2 (Ucn-2) has demonstrated cardioprotective actions against myocardial ischemia-reperfusion (I/R) injuries. Herein, we explored the protective role of Ucn-2 through microRNAs (miRNAs) post-transcriptional ...
    • Role of NOD1 in heart failure progression via regulation of Ca2+ handling 

      Val-Blasco, Almudena; Piedras, María José; Ruiz-Hurtado, Gema; Suárez-Fuentetaja, Natalia; Prieto, Patricia; González-Ramos, Silvia; Gómez-Hurtado, Nieves; Delgado, Carmen; Pereira, Laetitia; Benito, Gemma; Zaragoza, Carlos; Doménech, Nieves; Crespo-Leiro, María Generosa; Vázquez-Echeverri, Daniel; Núñez, Gabriel; López-Collazo, Eduardo; Boscá, Lisardo; Fernández-Velasco, María (Elsevier para American College of Cardiology Foundation, 2017-01-23)
    • Specialized proresolving mediators protect against experimental autoimmune myocarditis by modulating Ca2+ handling and NRF2 activation 

      Val-Blasco, Almudena; Prieto, Patricia; Íñigo Jaén, Rafael; Gil-Fernández, Marta; Pajares, Marta; Doménech, Nieves; Terrón, Verónica; Tamayo, María; Jorge, Inmaculada; Vázquez, Jesús; Bueno-Sen, Andrea; Vallejo-Cremades, María Teresa; Pombo-Otero, Jorge; Sánchez-García, Sergio; Ruiz-Hurtado, Gema; Gómez, Ana María; Zaragoza, Carlos; Crespo-Leiro, María Generosa; López-Collado, Eduardo; Cuadrado, Antonio; Delgado, Carmen; Boscá, Lisardo; Fernández-Velasco, María (Elsevier, 2022-05-18)
      [Abstract] Specialized proresolving mediators and, in particular, 5(S), (6)R, 7-trihydroxyheptanoic acid methyl ester (BML-111) emerge as new therapeutic tools to prevent cardiac dysfunction and deleterious cardiac damage ...